Peptic ulcer disease
Structured condition card with NCLEX priority cues and nursing action focus.
GI / Liver / Pancreasmedium priorityneeds review
Peptic ulcer disease
Etiology / Pathophysiology
- H. pylori infection, NSAID use, stress physiology, or excess acid injury.
- Mucosal barrier breakdown creates gastric or duodenal ulceration.
Medications
| Class | Why it matters |
|---|---|
| GI acid reducers | Promotes ulcer healing. |
| Antibiotics by class | Used for H. pylori regimens. |
Nursing actions
- Assess pain pattern, NSAID use, bleeding signs, and anemia symptoms.
- Teach avoiding NSAIDs/alcohol if instructed and completing H. pylori therapy.
- Escalate sudden severe abdominal pain or rigid abdomen.
Complications
- GI bleeding
- Perforation
- Gastric outlet obstruction
NCLEX cues
- Coffee-ground emesis or black tarry stool.
- Board-like abdomen can mean perforation.
Memory hooks
- Ulcer can bleed or perforate.
Labs / Diagnostics
- Trend assessment findings and ordered diagnostics; verify exact values with school source material.
Review notes
- Session-derived study seed. Verify against school materials, ATI/NCLEX review sources, current orders, and facility policy before relying on details.