Peptic ulcer disease
Structured condition card with NCLEX priority cues and nursing action focus.
GI / Liver / Pancreasmedium priorityneeds review
Peptic ulcer disease
Etiology / Pathophysiology
- H. pylori infection, NSAID use, stress physiology, or excess acid injury.
- Mucosal barrier breakdown creates gastric or duodenal ulceration.
Medications
| Class | Why it matters |
|---|---|
| GI acid reducers | Promotes ulcer healing. |
| Antibiotics by class | Used for H. pylori regimens. |
Signs / symptoms
- Coffee-ground emesis or black tarry stool.
- Board-like abdomen can mean perforation.
Nursing actions
- Assess pain pattern, NSAID use, bleeding signs, and anemia symptoms.
- Teach avoiding NSAIDs/alcohol if instructed and completing H. pylori therapy.
- Escalate sudden severe abdominal pain or rigid abdomen.
Complications
- GI bleeding
- Perforation
- Gastric outlet obstruction
NCLEX cues
- Coffee-ground emesis or black tarry stool.
- Board-like abdomen can mean perforation.
Memory hooks
- Ulcer can bleed or perforate.
Labs / Diagnostics
- Trend assessment findings and ordered diagnostics; verify exact values with school source material.
Review notes
- Session-derived study seed. Verify against school materials, ATI/NCLEX review sources, current orders, and facility policy before relying on details.
Cross references
Related cards are generated from the shared study map, not hand-built per page.
Source entitycondition card
Peptic ulcer disease
Condition card for Peptic ulcer disease: recognize the testable cue, prevent the complication, and choose the safest first nursing action.
Procedurecondition
Endoscopy
Scope procedure used to view or sample the GI tract.
Med classmedication safety
Antibiotics by class
Targets bacterial growth or cell structures; exact teaching depends on the antibiotic class.
Med classmedication safety
GI acid reducers
Reduces stomach acid so irritated tissue can heal and reflux symptoms decrease.