Myasthenia gravis
Structured condition card with NCLEX priority cues and nursing action focus.
NeuroAutoimmune / Genetichigh priorityneeds review
Myasthenia gravis
Etiology / Pathophysiology
- Autoimmune attack on acetylcholine receptors at the neuromuscular junction.
- Muscles weaken with use and improve with rest; respiratory muscles can fail in crisis.
Medications
| Class | Why it matters |
|---|---|
| Anticholinesterase agents | Improves neuromuscular transmission. |
Nursing actions
- Assess respiratory effort, swallowing, chewing fatigue, and ptosis.
- Schedule activities after medication peak when possible.
- Keep suction and airway support available for bulbar weakness.
Complications
- Myasthenic crisis
- Aspiration
- Respiratory failure
NCLEX cues
- Ptosis, diplopia, dysphagia, weakness worse later in day.
- Respiratory decline is priority.
Memory hooks
- MG muscles get tired; meals after meds.
Labs / Diagnostics
- Trend assessment findings and ordered diagnostics; verify exact values with school source material.
Review notes
- Session-derived study seed. Verify against school materials, ATI/NCLEX review sources, current orders, and facility policy before relying on details.