Myasthenia gravis
Structured condition card with NCLEX priority cues and nursing action focus.
NeuroAutoimmune / Genetichigh priorityneeds review
Myasthenia gravis
Etiology / Pathophysiology
- Autoimmune attack on acetylcholine receptors at the neuromuscular junction.
- Muscles weaken with use and improve with rest; respiratory muscles can fail in crisis.
Medications
| Class | Why it matters |
|---|---|
| Anticholinesterase agents | Improves neuromuscular transmission. |
Signs / symptoms
- Ptosis, diplopia, dysphagia, weakness worse later in day.
- Respiratory decline is priority.
Nursing actions
- Assess respiratory effort, swallowing, chewing fatigue, and ptosis.
- Schedule activities after medication peak when possible.
- Keep suction and airway support available for bulbar weakness.
Complications
- Myasthenic crisis
- Aspiration
- Respiratory failure
NCLEX cues
- Ptosis, diplopia, dysphagia, weakness worse later in day.
- Respiratory decline is priority.
Memory hooks
- MG muscles get tired; meals after meds.
Labs / Diagnostics
- Trend assessment findings and ordered diagnostics; verify exact values with school source material.
Review notes
- Session-derived study seed. Verify against school materials, ATI/NCLEX review sources, current orders, and facility policy before relying on details.
Cross references
Related cards are generated from the shared study map, not hand-built per page.
Source entitycondition card
Myasthenia gravis
Condition card for Myasthenia gravis: recognize the testable cue, prevent the complication, and choose the safest first nursing action.
Med classmedication safety
Anticholinesterase agents
Keeps acetylcholine active longer at the neuromuscular junction to improve muscle strength.